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Anti-cortisol

OBJECTIVE: RELAXING FOR A HEALTHIER SKIN

End of holidays, back to school, work overload, change of season, worries … the reasons for stressing are not lacking, and the effects of stress on the skin are immediately noticeable. The skin is not only a reflection of our health, but also of our moods.

STRESS AND SKIN

Cortisol (the stress hormone) is secreted during stress peaks. It boosts the body to help us overcome unusual situations.
However, in chronic stress (active life, anxiety …) the constant production of cortisol disrupts the structures and metabolisms of the skin.
The visible and thus aesthetic repercussions do not take long to appear: brittle hair, dandruff, rapid variation of weight, thinner skin, dull, dry, loss of elasticity …
In the medium term, chronic stress may also trigger skin diseases such as certain dermatoses.
And as if that was not enough, cortisol slows the regeneration and healing abilities of the skin.
In the long-term, cortisol can therefore be responsible for the installation of morphological variations, which are also a source of stress: A real vicious circle.

CONSUMERS’ EXPECTATIONS

Feeling good
 
  • Decreasing the visible signs of skin fatigue
  • Preventing weight gain due to stress
  • Finding solutions for skin problems: reducing redness, optimizing regeneration, preventing and treating dermatitis

EXSYMOL’S ANSWER

To meet these expectations, Exsymol has developed a 3-step anti-stress response, which combines the effectiveness of SILANOLS and the neurocosmetic technology.

Step 1: Protecting the skin

Stress may cause:

  • A slow down of the skin’s regeneration abilities
  • An increase of the sensitivity of the epidermis.
  • Dehydration
  • Transparency of the skin
  • A blurred complexion

Keratinocytes have filopods, cellular outgrowths, which role is key in the capacity of motricity cells. The filopodia are also the seat of the hyalurosome that controls the production of hyaluronic acid.
Cortisol specifically targets filopods and is responsible for their atrophy. Keratinocytes therefore lose their mobility and their ability to produce HA decreases. The skin is less hydrated, less protected and its capacity for regeneration (healing) is largely affected.

Epidermosil

Step 2: Normalizing skin cell metabolisms

Oily skin

Stress induces an increase in sebum (in green) production by sebocytes. Cortisol is responsible for this overproduction.

Epidermosil

Regeneration (scar healing) slow down

The prevention or the treatment of dermatoses requires optimal regenerative capacities. It is the same for the healing process. Keratinocyte mobility and proliferation rate are essential characteristics to preserve.

Epidermosil

Step 3: Take back control of its forms

Stress also induces a slowi down of the cell metabolism together with a collapse of both the dermis and the hypodermis.

  • Loss of firmness and elasticity
  • Fat accumulation

Adipose tissue is very sensitive to cortisol. In times of stress, weight gain is often observed.

Glutrapeptide

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